The diagnosis of LGV consists of detecting the nucleic acids of C. These clinical signs and symptoms require a differential diagnosis with other causes of proctitis, such as inflammatory bowel disease. In Europe, this sexually transmitted infection manifests as proctitis with rectal pain, tenesmus, anorectal bleeding, and constipation, which can develop into abscesses, fistulae, and rectal stenosis if not treated in the initial phases of the disease. Classical presentation of LGV, typical of Asia and the Americas, is divided into 3 phases: the first phase is characterized by a painless sore at the inoculation site, which appears between 3 and 30 days after contact the second phase involves the appearance of painful enlarged lymph nodes and the final phase involves lymphedema and elephantiasis due to the irreversible destruction of the lymphatic system if left untreated. The clinical presentation of LGV varies depending on the geographic location of the cases. 1,3–6 The growing incidence of this disease in Spain led it to be included as a disease subject to mandatory reporting, individualized since 2015. This infection was rare in Europe until 2003, when the incidence of cases of the disease began to increase, mainly affecting men who have sex with HIV-positive men. 1–4 It is transmitted via vaginal, anal, or oral sexual contact. LGV is a sexually transmitted infection caused by serovars L1, L2, and 元 of Chlamydia trachomatis, which is endemic in tropical countries of Asia and the Americas. In light of the diagnosis of lymphogranuloma venereum (LGV), the patient was treated with doxycycline at a dosage of 100 mg every 12 hours for 21 days, with complete remission of clinical symptoms.
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